Same old: 2008

Thursday 13 November 2008

Cannabislike drugs may hold key to treating pain while bypassing the brain.

Cannabislike drugs may hold key to treating pain while bypassing the brain.

JAMA. 2008 Nov 5;300(17):1987

Authors: Hampton T

PMID: 18984881 [PubMed - indexed for MEDLINE]

View Original Article

Monday 10 November 2008

No major role of common SV2A variation for predisposition or levetiracetam response in epilepsy.

No major role of common SV2A variation for predisposition or levetiracetam response in epilepsy.

Epilepsy Res. 2008 Oct 30;

Authors: Lynch JM, Tate SK, Kinirons P, Weale ME, Cavalleri GL, Depondt C, Murphy K, O'Rourke D, Doherty CP, Shianna KV, Wood NW, Sander JW, Delanty N, Goldstein DB, Sisodiya SM

Levetiracetam (LEV), a newer antiepileptic drug (AED) useful for several epilepsy syndromes, binds to SV2A. Identifying genetic variants that influence response to LEV may allow more tailored use of LEV. Obvious candidate genes are SV2A, SV2B and SV2C, which encode the only known binding site, synaptic vesicle protein 2 (SV2), with LEV binding to the SV2A isoform. SV2A is an essential protein as homozygous SV2A knockout mice appear normal at birth but fail to grow, experience severe seizures and die by 3 weeks. We addressed characterising AED response issues in pharmacogenetics and whether variation in these genes associates with response to LEV in two independent cohorts with epilepsy. We also investigated whether variation in these three genes associated with epilepsy predisposition in two larger cohorts of patients with various epilepsy phenotypes. Common genetic variation in SV2A, encoding the actual binding site of LEV, was fully represented in this study whereas SV2B and SV2C were not fully covered. None of the polymorphisms tested in SV2A, SV2B or SV2C influence LEV response or predisposition to epilepsy. We found no association between genetic variation in SV2A, SV2B or SV2C and response to LEV or epilepsy predisposition. We suggest this study design may be used in future pharmacogenetic work examining AED or LEV efficacy. However, different study designs would be needed to examine common variation with minor effect sizes, or rare variation, influencing AED or LEV response or epilepsy predisposition.

PMID: 18977120 [PubMed - as supplied by publisher]

View Original Article

Pro-drugs for indirect cannabinoids as therapeutic agents.

Pro-drugs for indirect cannabinoids as therapeutic agents.

Curr Drug Deliv. 2008 Oct;5(4):243-7

Authors: Ashton J

Medicinal cannabis, cannabis extracts, and other cannabinoids are currently in use or under clinical trial investigation for the control of nausea, emesis and wasting in patients undergoing chemotherapy, the control of neuropathic pain and arthritic pain, and the control of the symptoms of multiple sclerosis. The further development of medicinal cannabinoids has been challenged with problems. These include the psychoactivity of cannabinoid CB1 receptor agonists and the lack of availability of highly selective cannabinoid receptor full agonists (for the CB1 or CB2 receptor), as well as problems of pharmacokinetics. Global activation of cannabinoid receptors is usually undesirable, and so enhancement of local endocannabinoid receptor activity with indirect cannabimimetics is an attractive strategy for therapeutic modulation of the endocannabinoid system. However, existing drugs of this type tend to be metabolized by the same enzymes as their target endocannabinoids and are not yet available in a form that is clinically useful. A potential solution to these problems may now have been suggested by the discovery that paracetamol (acetaminophen) exerts its analgesic (and probably anti-pyretic) effects by its degradation into an anandamide (an endocannabinoid) reuptake inhibitor (AM404) within the body, thus classifying it as pro-drug for an indirect cannabimimetic. Given the proven efficacy and safety of paracetamol, the challenge now is to develop related drugs, or entirely different substrates, into pro-drug indirect cannabimimetics with a similar safety profile to paracetamol but at high effective dose titrations.

PMID: 18855592 [PubMed - in process]

View Original Article

Evaluation of interactions between cannabinoid compounds and diazepam in electroshock-induced seizure model in mice.

Evaluation of interactions between cannabinoid compounds and diazepam in electroshock-induced seizure model in mice.

J Neural Transm. 2008 Jun 25;

Authors: Naderi N, Aziz Ahari F, Shafaghi B, Hosseini Najarkolaei A, Motamedi F

Several studies have shown that cannabinoids have anticonvulsant properties that are mediated through activation of the cannabinoid CB1 receptors. In addition, endogenous cannabinoid compounds (endocannabinoids) regulate synaptic transmission and dampen seizure activity via activation of the same receptors. The aim of this study was to evaluate the possible interactions between antiepileptic effects of cannabinoid compounds and diazepam using electroshock-induced model of seizure in mice. Electroconvulsions were produced by means of an alternating current (ear-clip electrodes, fixed current intensity 35 mA, stimulus duration 0.2 s) and tonic hindlimb extension was taken as the endpoint. All experiments were performed on groups of ten mice and the number of animals who did not display seizure reported as percent protection. Intraperitoneal (i.p.) administration of diazepam (0.25-2 mg/kg) and CB1 receptor agonist WIN55212-2 (0.5-4 mg/kg) dose dependently produced an antiepileptic effect evaluated in terms of increased percentage of protection against electroshock-induced seizure. Logistic regression analysis indicated synergistic interactions in anticonvulsant action after co-administration of diazepam and WIN55212-2 in fixed-ratio combination of 3:1 (diazepam:WIN55212-2), while an additive effect was resulted after co-administration of 1:1 and 1:3 fixed-ratio combinations. Administration of various doses of the endocannabinoid reuptake inhibitor, AM404, did not produce any effect on electroshock-induced seizure. Moreover, co-administration of AM404 and diazepam did not produce significant interaction in antiepileptic properties of these compounds. Administration of the fatty acid amide hydrolase inhibitor, URB597, produced significant antiepileptic effect. Co-administration of URB597 and diazepam led to an antagonistic interaction in protection against shock-induced seizure. Co-administration of different doses of the cannabinoid CB1 receptor antagonist, AM251 did not alter the antiepileptic effect of diazepam in the electroshock-induced seizure test. These results demonstrate that endocannabinoid system participates in the modulation of seizure and combination of small doses of exogenous CB1 receptor agonists with diazepam may have effective consequences in seizure control. Furthermore, inhibiting the endocannabinoid degradation could be more efficacious in modulating seizure than preventing their uptake. This study also suggests that the effects of cannabinoids on epilepsy depend on the relative cannabinoid responsiveness of GABAergic and glutamatergic neurotransmission. While, the antiepileptic effects of cannabinoid compounds are likely by affecting excitatory glutamate neurotransmission, the antagonistic interaction between cannabinoid compounds and diazepam to protect seizure is due to the cannabinoid action on inhibitory GABAergic system.

PMID: 18575801 [PubMed - as supplied by publisher]

View Original Article

Elevation of pentylenetetrazole-induced seizure threshold in cholestatic mice: interaction between opioid and cannabinoid systems.

Elevation of pentylenetetrazole-induced seizure threshold in cholestatic mice: interaction between opioid and cannabinoid systems.

J Gastroenterol Hepatol. 2008 Jul;23(7 Pt 2):e251-7

Authors: Shafaroodi H, Ghasemi M, Dehpour AR

BACKGROUND AND AIM: Several studies have reported that endogenous opioid and cannabinoid systems may be involved in some pathophysiological changes occurring in cholestatic liver disease. It is well known that endogenous opioids and cannabinoids alter the susceptibility of experimental animals to different models of seizure. METHODS: The alterations in pentylenetetrazole-induced clonic seizure thresholds were evaluated from 1 to 6 days after bile duct ligation in mice. Whether the pretreatment of cholestatic mice with different doses of opioid receptor antagonist naltrexone or cannabinoid CB(1) receptor antagonist AM251 (AM251) would have changed the clonic seizure threshold was also examined. RESULTS: Although the clonic seizure threshold was similar between sham-operated and unoperated mice, there was a time-dependent increase in the threshold in cholestatic mice, reaching a peak on day 3 after bile duct ligation and declining partially after day 4. Chronic pretreatment with naltrexone (2, 5, and 10 mg/kg) reversed the increased threshold in cholestatic mice on day 3 after operation in a dose-dependent manner with the highest doses used restoring the threshold to that of the control animals. A similar reversal of the increased threshold was observed after acute (0.5, 0.75, and 1 mg/kg) or chronic (0.5 mg/kg for 4 days) pretreatment with AM251. Moreover, concurrent administration of doses of AM251 and naltrexone that each separately induced a partial reversal of increased seizure threshold in cholestasis caused a complete restoring of the threshold to the control level. CONCLUSIONS: Both opioid and cannabinoid CB(1) receptors may be involved in the dramatic increase in pentylenetetrazole-induced seizure threshold in cholestasis.

PMID: 17764531 [PubMed - indexed for MEDLINE]

View Original Article

Effects of levetiracetam on generalized discharges monitored with ambulatory EEG in epileptic patients.

Effects of levetiracetam on generalized discharges monitored with ambulatory EEG in epileptic patients.

Seizure. 2008 Oct 1;

Authors: Pro S, Vicenzini E, Pulitano P, Voti PL, Zarabla A, Randi F, Mecarelli O

PURPOSE: Quantitative analysis of epileptiform discharges (EDs) before and after the initiation of an antiepileptic treatment is a useful tool to objectively documentate the efficacy of an antiepileptic drug (AED). Aim of this study was to evaluate the effect of levetiracetam (LEV) on EDs, monitored with ambulatory EEG (A/EEG), in a limited series of patients with generalized epilepsy. METHODS: We performed 24h A/EEG recording in basal condition and at follow-up after LEV therapy in 21 adult epileptic patients. Eleven received LEV as monotherapy and 10 as add-on. For each patient we quantified total epileptic activity considering the following parameters: total number, total duration, maximal duration and median duration of EDs. Self-reported information on the effect of LEV on clinical seizures was also collected, to determine the electro-clinical correlation. RESULTS: A high variability of the response to LEV was observed in the monotherapy group, without statistical differences for all the parameters investigated. A significant reduction of the total number of seizures (113.6 vs. 41.2; p=.01) was observed in patients in add-on therapy. The modifications of epileptiform EEG abnormalities did not necessarily correlate with the self-reported clinical impressions. DISCUSSION: The quantification of EDs monitored by A/EEG provides a useful objective support for evaluating the neurophysiologic profile and the real efficacy of an antiepileptic treatment. In our patients LEV was able to significantly reduce the EDs only in add-on therapy. Further larger studies are necessary to clarify the effects of LEV on electro-clinical features of generalized epilepsy.

PMID: 18835193 [PubMed - as supplied by publisher]

View Original Article

Distribution of the olfactory fibre input into the olfactory tubercle of the in vitro isolated guinea pig brain.

Distribution of the olfactory fibre input into the olfactory tubercle of the in vitro isolated guinea pig brain.

J Neurophysiol. 2008 Oct 15;

Authors: Carriero G, Uva L, Gnatkovsky V, de Curtis M

The olfactory tubercle (OT) is a cortical component of the olfactory system involved in reward mechanisms of drug abuse. This region covers an extensive part of the rostral ventral cerebrum and is relatively poorly investigated. The intrinsic network interactions evoked by olfactory input are here analysed in the OT of the in vitro isolated guinea pig brain by means of field potential analysis and optical imaging of voltage-sensitive signals. Stimulation of the lateral olfactory tract induces a monosynaptic response that progressively decreases in amplitude from lateral to medial. The monosynaptic input induces a disynaptic response that is proportionally larger in the medial portion of the OT. Direct stimulation of the piriform cortex and subsequent lesion of this pathway demonstrated the existence of a prominent associative projection from the anterior part of the piriform cortex to the lateral part of the OT. Optical and electrophysiological recordings of the signals evoked by stimulation of the olfactory tract during arterial perfusion with the voltage-sensitive dye di-2-ANEPEQ confirmed the pattern of distribution of the mono and disynaptic responses in the OT. Finally, current source density analysis of laminar profiles recorded with 16-channel silicon probes confirmed that the monosynaptic and disynaptic potentials localize in the most superficial and the deep portions of the plexiform layer I, as suggested by previous reports. This study sets the standard for further analysis of the modulation of network properties in this largely unexplored brain region.

PMID: 18922946 [PubMed - as supplied by publisher]

View Original Article

Pavlov's moth: olfactory learning and spike timing-dependent plasticity.

Pavlov's moth: olfactory learning and spike timing-dependent plasticity.

Nat Neurosci. 2008 Oct;11(10):1126-7

Authors: Meeks JP, Holy TE

PMID: 18818593 [PubMed - indexed for MEDLINE]

View Original Article

Differential potentiation of early and late components evoked in olfactory cortex by stimulation of cortical association fibers.

Differential potentiation of early and late components evoked in olfactory cortex by stimulation of cortical association fibers.

Brain Res. 2008 Oct 17;

Authors: Stripling JS, Galupo MP

The present study examined in detail the development and decay of potentiation induced in vivo by repeated high-frequency stimulation of cortical association fibers (AF) in piriform cortex (PC). Male Long-Evans rats with chronically-implanted stimulating and recording electrodes were administered potentiating AF stimulation (thirty 10-pulse 100-Hz trains) on 8 consecutive days, followed by a ninth administration after an 8-day layoff. The time course of potentiation was monitored by local field potentials evoked in the PC and olfactory bulb (OB) by 0.1 Hz single-pulse AF test stimulation before, during, and following each potentiating treatment. AF test stimulation evoked two distinct components in the PC, an early component (EC) and a late component (LC). High-frequency AF stimulation produced potentiation of each component, but with very different characteristics. EC potentiation consisted of a brief augmentation during each bout of potentiating stimulation that persisted <2 min after the last high-frequency train and showed no cumulative effects following repeated induction across days. In contrast, LC potentiation developed gradually, requiring several daily potentiation treatments to reach maximum amplitude, and decayed more slowly each time it was induced. Furthermore, LC potentiation persisted in latent form for at least 8 days following its apparent decay and could be reinstated by repeated test stimulation that was without effect at the beginning of the experiment. Potentiation in the OB resembled LC potentiation in its characteristics, but with less latent potentiation. These results indicate that the potentiation reported here is distinctly different from the long-term potentiation previously demonstrated in vitro in the PC, and suggest that this potentiation represents an increase in excitability within the cortical association fiber system that can be stored in latent form and retrieved at a later time. These characteristics make this potentiation a suitable candidate for participation in long-term functional changes within olfactory cortex.

PMID: 18955033 [PubMed - as supplied by publisher]

View Original Article

Vote on Graphs « GraphJam: Music and Pop Culture in Charts and Graphs. Let us explain them.

Wednesday 5 November 2008

Why no c*nt and paste for the iPhone? • The Register

Thursday 16 October 2008

Fukitol: The New Miracle Drug | Nick Lewis: The Blog

Monday 13 October 2008

MDA7: a novel selective agonist for CB(2) receptor...[Br J Pharmacol. 2008] - PubMed Result

Friday 10 October 2008

Sad Guys on Trading Floors - Jazzhands will not save us now, my friend.

Sad Guys on Trading Floors - Ok, yeah, I’m holding down the reset button down....

Friday 3 October 2008

Psychological and social sequelae of cannabis and ...[Lancet. 2004] - PubMed Result

Facebook | Home

Wednesday 1 October 2008

The role of piriform cortex adenosine A1 receptors on hippocampal kindling.

The role of piriform cortex adenosine A1 receptors on hippocampal kindling.

Can J Neurol Sci. 2008 May;35(2):226-31

Authors: Namvar S, Mirnajafi-Zadeh J, Fathollahi Y, Zeraati M

INTRODUCTION: The hippocampus and piriform cortex have a critical role in seizure propagation. In this study, the role of adenosine A1 receptors of piriform cortex on CA1 hippocampal kindled seizures was studied in rats. METHODS: Animals were implanted with a tripolar electrode in the right hippocampal CA1 region and two guide cannulae in the left and right piriform cortex. They were kindled by daily electrical stimulation of hippocampus. In fully kindled rats, N6- cyclohexyladenosine (CHA; a selective adenosine A1 receptors agonist) and 1,3-dimethyl-8-cyclopenthylxanthine (CPT a selective adenosine A1 receptor antagonist) were microinfused into the piriform cortex. The animals were stimulated at 5, 15 and 90 minutes (min) after drug injection. RESULTS: Obtained data showed that CHA (10 and 100 microM) reduced afterdischarge duration, stage 5 seizure duration, and total seizure duration at 5 and 15 min after drug injection. There was no significant change in latency to stage 4 seizure. CPT at concentration of 20 microM increased afterdischarge duration, stage 5 seizure duration, and total seizure duration and decreased latency to stage 4 seizure at 5 and 15 min post injection. Pretreatment of rats with CPT (10 microM), 5 min before CHA (100 microM), reduced the effect of CHA on seizure parameters. CONCLUSION: These results suggested that activity of adenosine A1 receptors in the piriform cortex has an anticonvulsant effect on kindled seizures resulting from electrical stimulation of the CA1 region of the hippocampus.

PMID: 18574939 [PubMed - in process]

View Original Article

Age- and region-dependent patterns of Ca(2+) accumulations following status epilepticus.

Age- and region-dependent patterns of Ca(2+) accumulations following status epilepticus.

Int J Dev Neurosci. 2008 Jul 17;

Authors: Friedman LK, Saghyan A, Peinado A, Keesey R

Elevated Ca(2+) concentrations have been implicated in cell death mechanisms following seizures, however, the age and brain region of intracellular Ca(2+) accumulations [Ca(2+)](i), may influence whether or not they are toxic. Therefore, we examined regional accumulations of (45)Ca(2+) by autoradiography from rats of several developmental stages (P14, P21, P30 and P60) at 5, 14, and 24h after status epilepticus. To determine whether the uptake was intracellular, Ca(2+) was also assessed in hippocampal slices with the dye indicator, Fura 2 AM at P14. Control animals accumulated low homogeneous levels of (45)Ca(2+); however, highly specific and age-dependent patterns of (45)Ca(2+) uptake were observed at 5h. (45)Ca(2+) accumulations were predominant in dorsal hippocampal regions, CA1/CA2/CA3a, in P14 and P21 rats and in CA3a and CA3c neurons of P30 and P60 rats. Selective midline and amygdala nuclei were marked at P14 but not at P21 and limbic accumulations recurred with maturation that were extensive at P30 and even more so at P60. At 14h, P14 and P21 rats had no persistent accumulations whereas P30 and P60 rats showed persistent uptake patterns within selective amygdala, thalamic and hypothalamic nuclei, and other limbic cortical regions that continued to differ at these ages. For example, piriform cortex accumulation was highest at P60. Fura 2AM imaging at P14 confirmed that Ca(2+) rises were intracellular and occurred in both vulnerable and invulnerable regions of the hippocampus, such as CA2 pyramidal and dentate granule cells. Silver impregnation showed predominant CA1 injury at P20 and P30 but CA3 injury at P60 whereas little or no injury was found in extrahippocampal structures at P14 and P20 but was modest at P30 and maximal at P60. Thus, at young ages there was an apparent dissociation between high (45)Ca(2+) accumulations and neurotoxicity whereas in adults a closer relationship was observed, particularly in the extrahippocampal structures.

PMID: 18687397 [PubMed - as supplied by publisher]

View Original Article

GABAergic circuits control input-spike coupling in the piriform cortex.

GABAergic circuits control input-spike coupling in the piriform cortex.

J Neurosci. 2008 Aug 27;28(35):8851-9

Authors: Luna VM, Schoppa NE

Odor coding in mammals is widely believed to involve synchronized gamma frequency (30-70 Hz) oscillations in the first processing structure, the olfactory bulb. How such inputs are read in downstream cortical structures however is not known. Here we used patch-clamp recordings in rat piriform cortex slices to examine cellular mechanisms that shape how the cortex integrates inputs from bulb mitral cells. Electrical stimulation of mitral cell axons in the lateral olfactory tract (LOT) resulted in excitation of pyramidal cells (PCs), which was followed approximately 10 ms later by inhibition that was highly reproducible between trials in its onset time. This inhibition was somatic in origin and appeared to be driven through a feedforward mechanism, wherein GABAergic interneurons were directly excited by mitral cell axons. The precise inhibition affected action potential firing in PCs in two distinct ways. First, by abruptly terminating PC excitation, it limited the PC response to each EPSP to exactly one, precisely timed action potential. In addition, inhibition limited the summation of EPSPs across time, such that PCs fired action potentials in strong preference for synchronized inputs arriving in a time window of <5 ms. Both mechanisms would help ensure that PCs respond faithfully and selectively to mitral cell inputs arriving as a synchronized gamma frequency pattern.

PMID: 18753387 [PubMed - in process]

View Original Article

Wednesday 6 August 2008

Google Image Result for http://bp2.blogger.com/_Se19XAPwVi8/RnBpY4DNShI/AAAAAAAABPM/blIEFrJgP7k/s400/rimonabant.jpg

View Original Article

Blogged with the Flock Browser

Thursday 29 May 2008

Fedora: Re: Logitech Webcam(USB) New Errors Posted

Re: Logitech Webcam(USB) New Errors PostedFrom: Murali Parth (muralinux48_at_yahoo.com)Date: 07/05/04# Next message: Wolfgang Gill: "Re: Logitech Webcam(USB) New Errors Posted" * Previous message: Jeff Allison: "Re: Correct driver for VIA KM400 onboard graphics with FC2." * Next in thread: Wolfgang Gill: "Re: Logitech Webcam(USB) New Errors Posted" * Reply: Wolfgang Gill: "Re: Logitech Webcam(USB) New Errors Posted" * Messages sorted by: [ date ] [ thread ] [ subject ] [ author ] Date: Sun, 4 Jul 2004 20:32:24 -0700 (PDT)To: Redhat Fedora Hello List !Sorry to bother you again, but with fresher errors.Idownloaded the driver for Logitech QUICKCAM EXPRESSfrom the site : http://qce-ga.sourceforge.net.Thenafter extracting I ran the script "quickcam.sh" andthe following are the messages I got.It seems that theproblem lies with detection of the device.When I doan ls -l /dev/video0 and it displays the major,minornos. as 81 & 0 and /dev/video as 81 & 1.I removed thefile hwconf and ran "kudzu".It detects the USBcontroller as "desc: "VIA Technologies|VT82xxxxx UHCIUSB 1.1 Controller" .Now the result of quickcam.shfollows :'Now everything should be well and the drivercompiled.Let's then try actually loading the fresh driver andtestingif it works.Press Ctrl+C to quit, Enter to continue --->To load the driver, I need to know the root password.=== Entering root mode === I will now try to enable the SysRq key.If your computer crashes, you can try pressing: Alt + SysRq + S: Emergency Sync (writeeverything on hard disk) Alt + SysRq + U: Unmount all harddisks Alt + SysRq + B: Reboot system immediatelyPress Ctrl+C to quit, Enter to continue --->Now I finally will try to load the module.If you're unlucky, your computer might crash rightnow!!!!Consider long if you really want to continue.Press Ctrl+C to quit, Enter to continue --->You decided to do it, here we go...=== Leaving root mode ===The driver detected the following supported cameras:[!] No cameras detected.Try unloading and reloading the driver manually with rmmod quickcam; insmod ./quickcam.ko debug=-1and then checking whether there are any messagesindicatingproblems with command dmesgWARNING: I'll try to continue anyway, but thisprobably will fail.Press Ctrl+C to quit, Enter to continue --->I will be using , if there are more cameras I'll nottest them.Press Ctrl+C to quit, Enter to continue --->Testing if is correct.ls: : No such file or directoryls: : No such file or directory./quickcam.sh: line 547: [: too many argumentsls: : No such file or directoryls: : No such file or directory[!] major number is .Usually it should be 81, so there are problems ahead.WARNING: I'll try to continue anyway, but thisprobably will fail.Press Ctrl+C to quit, Enter to continue ---> Right now driver is loaded and ready to run.If you like, you can quit now and start using thecamera -you have good chances that it works, if no problemswere detected.If you have X Window System running and xawtvinstalled,I can now run it automatically for you.You will then also have opportunity to install thedriver permanently.Press Ctrl+C to quit, Enter to continue --->Launching xawtv (press q on xawtv window to quitit)... xawtv -noscale -noxv -c ""This is xawtv-3.92, running on Linux/i686(2.6.5-1.358)v4l-conf: option requires an argument -- cusage: v4l-conf [ options ] options: -q quiet -d X11 Display [:0.0] -c video device [/dev/video0] -b displays color depth is bpp -s shift display by bytes -f query frame buffer device for info -a set framebuffer address to (in hex, root only, successfulautodetect will overwrite this address) -1 force v4l API -2 force v4l2 APIv4l-conf had some trouble, trying to continue anywayv4l2: open : No such file or directoryv4l2: open : No such file or directoryv4l: open : No such file or directoryno video grabber device available Well, did it work, did you get a picture?If you did, you might now want to install the driverpermanently. Just proceed to do that...Press Ctrl+C to quit, Enter to continue --->Just an extra warning: the driver (quickcam.ko) andthe utility (qcset) will be now copied into systemdirectories. If you have already other versions,they will be overwritten. Verify by giving rootpassword.=== Entering root mode ===/usr/bin/install -c -D -m 644 quickcam.ko/lib/modules/2.6.5-1.358/misc/quickcam.ko/usr/bin/install -c -D -m 755 qcset/usr/local/bin/qcset/sbin/depmod -a=== Leaving root mode ===Hopefully the driver is now installed and can beloadedwith command modprobe quickcamas root. You can put this command into some startupscript to do it always automatically at boot.The exact location depends on distribution, and thisscript is yet too dumb to do this automatically.Press Ctrl+C to quit, Enter to continue --->Goodbye... 'Then I did the following :[root@muralinux qc-usb-0.6.0]# modprobe quickcam[root@muralinux qc-usb-0.6.0]# lsmodModule Size Used byquickcam 59060 0ohci_hcd 14748 0uhci_hcd 23708 0tuner 15380 0bttv 129292 0video_buf 14468 1 bttvi2c_algo_bit 7304 1 bttvv4l2_common 4864 1 bttvbtcx_risc 3592 1 bttvi2c_core 16388 3tuner,bttv,i2c_algo_bitvideodev 6656 2 quickcam,bttv---------------------------------------------Could anyone help me to provide clue to unravellingthis problem ? Thanks.Murali__________________________________________________Do You Yahoo!?Tired of spam? Yahoo! Mail has the best spam protection aroundhttp://mail.yahoo.com-- fedora-list mailing listfedora-list@redhat.comTo unsubscribe: http://www.redhat.com/mailman/listinfo/fedora-list# Next message: Wolfgang Gill: "Re: Logitech Webcam(USB) New Errors Posted" * Previous message: Jeff Allison: "Re: Correct driver for VIA KM400 onboard graphics with FC2." * Next in thread: Wolfgang Gill: "Re: Logitech Webcam(USB) New Errors Posted" * Reply: Wolfgang Gill: "Re: Logitech Webcam(USB) New Errors Posted" * Messages sorted by: [ date ] [ thread ] [ subject ] [ author ] Relevant Pages * Re: [SLE] Missing 3D (was Compiz issues.) ... This is driver file location in above package ... post-uninstall script that will call tiny-nvidia-installer. ... Strange that it didn't offered to prepare existing kernel sources first. ... It can be that Smart did just what is programmed to do, install rpm, run ... (SuSE) * Re: vbs to configure printer to remote machine ... Description The printer driver is unknown. ... Win32 error code 1797 ... copy the files you want to install into this directory. ... Microsoft Windows Script Host Version 5.6 ... (microsoft.public.windowsxp.print_fax) * Re: Adding IP printer and install driver via script ... The driver has to be installed using the setup program from HP. ... an inf file in \Windows\inf you might be able to install it with the "Add ... I figured out how to creat the standard TCP/IP port but I'm ... > This is the error that I get when I run the script I created... ... (microsoft.public.windows.server.scripting) * Re: a script to add a new printer ... You install first the port and the driver, ... >> with the MS Windows 2003 Server Resource Kit from a script. ... (microsoft.public.scripting.wsh) * Re: apsfilter question/problem ... I don't have a fix to your apsfilter issue, but if you you are interested in another possible printing solution... ... that mentioned that driver, 10) PPA printer, so I chose that. ... the apsfilter setup script found it & didn't complain. ... Do I need to install another more complete version? ... (freebsd-questions) We are proud to have Web Hosting and Rack Housing from 9 Net Avenue Deutschland.(09)derkeiler.com * Security * UNIX * Linux * Coding * Usenet * Mailing-Lists * Newsgroups * About * Privacy * Search * ImprintPath: linux.derkeiler.com > Mailing-Lists > Fedora > 2004-07 Print as PDF RSS Feed

Fedora: Re: Logitech Webcam(USB) New Errors Posted

Blogged with the Flock Browser

Thursday 22 May 2008

Linux Kernel Documentation :: video4linux : CARDLIST.bttv

About Kernel Documentation Linux Kernel Contact Linux Resources Linux BlogDocumentation / video4linux / CARDLIST.bttvBased on kernel version 2.6.25. Page generated on 2008-04-18 21:22 EST.1 0 -> *** UNKNOWN/GENERIC ***2 1 -> MIRO PCTV3 2 -> Hauppauge (bt848)4 3 -> STB, Gateway P/N 6000699 (bt848)5 4 -> Intel Create and Share PCI/ Smart Video Recorder III6 5 -> Diamond DTV20007 6 -> AVerMedia TVPhone8 7 -> MATRIX-Vision MV-Delta9 8 -> Lifeview FlyVideo II (Bt848) LR26 / MAXI TV Video PCI2 LR2610 9 -> IMS/IXmicro TurboTV11 10 -> Hauppauge (bt878) [0070:13eb,0070:3900,2636:10b4]12 11 -> MIRO PCTV pro13 12 -> ADS Technologies Channel Surfer TV (bt848)14 13 -> AVerMedia TVCapture 98 [1461:0002,1461:0004,1461:0300]15 14 -> Aimslab Video Highway Xtreme (VHX)16 15 -> Zoltrix TV-Max [a1a0:a0fc]17 16 -> Prolink Pixelview PlayTV (bt878)18 17 -> Leadtek WinView 60119 18 -> AVEC Intercapture20 19 -> Lifeview FlyVideo II EZ /FlyKit LR38 Bt848 (capture only)21 20 -> CEI Raffles Card22 21 -> Lifeview FlyVideo 98/ Lucky Star Image World ConferenceTV LR5023 22 -> Askey CPH050/ Phoebe Tv Master + FM [14ff:3002]24 23 -> Modular Technology MM201/MM202/MM205/MM210/MM215 PCTV, bt878 [14c7:0101]25 24 -> Askey CPH05X/06X (bt878) [many vendors] [144f:3002,144f:3005,144f:5000,14ff:3000]26 25 -> Terratec TerraTV+ Version 1.0 (Bt848)/ Terra TValue Version 1.0/ Vobis TV-Boostar27 26 -> Hauppauge WinCam newer (bt878)28 27 -> Lifeview FlyVideo 98/ MAXI TV Video PCI2 LR5029 28 -> Terratec TerraTV+ Version 1.1 (bt878) [153b:1127,1852:1852]30 29 -> Imagenation PXC200 [1295:200a]31 30 -> Lifeview FlyVideo 98 LR50 [1f7f:1850]32 31 -> Formac iProTV, Formac ProTV I (bt848)33 32 -> Intel Create and Share PCI/ Smart Video Recorder III34 33 -> Terratec TerraTValue Version Bt878 [153b:1117,153b:1118,153b:1119,153b:111a,153b:1134,153b:5018]35 34 -> Leadtek WinFast 2000/ WinFast 2000 XP [107d:6606,107d:6609,6606:217d,f6ff:fff6]36 35 -> Lifeview FlyVideo 98 LR50 / Chronos Video Shuttle II [1851:1850,1851:a050]37 36 -> Lifeview FlyVideo 98FM LR50 / Typhoon TView TV/FM Tuner [1852:1852]38 37 -> Prolink PixelView PlayTV pro39 38 -> Askey CPH06X TView99 [144f:3000,144f:a005,a04f:a0fc]40 39 -> Pinnacle PCTV Studio/Rave [11bd:0012,bd11:1200,bd11:ff00,11bd:ff12]41 40 -> STB TV PCI FM, Gateway P/N 6000704 (bt878), 3Dfx VoodooTV 100 [10b4:2636,10b4:2645,121a:3060]42 41 -> AVerMedia TVPhone 98 [1461:0001,1461:0003]43 42 -> ProVideo PV951 [aa0c:146c]44 43 -> Little OnAir TV45 44 -> Sigma TVII-FM46 45 -> MATRIX-Vision MV-Delta 247 46 -> Zoltrix Genie TV/FM [15b0:4000,15b0:400a,15b0:400d,15b0:4010,15b0:4016]48 47 -> Terratec TV/Radio+ [153b:1123]49 48 -> Askey CPH03x/ Dynalink Magic TView50 49 -> IODATA GV-BCTV3/PCI [10fc:4020]51 50 -> Prolink PV-BT878P+4E / PixelView PlayTV PAK / Lenco MXTV-9578 CP52 51 -> Eagle Wireless Capricorn2 (bt878A)53 52 -> Pinnacle PCTV Studio Pro54 53 -> Typhoon TView RDS + FM Stereo / KNC1 TV Station RDS55 54 -> Lifeview FlyVideo 2000 /FlyVideo A2/ Lifetec LT 9415 TV [LR90]56 55 -> Askey CPH031/ BESTBUY Easy TV57 56 -> Lifeview FlyVideo 98FM LR50 [a051:41a0]58 57 -> GrandTec 'Grand Video Capture' (Bt848) [4344:4142]59 58 -> Askey CPH060/ Phoebe TV Master Only (No FM)60 59 -> Askey CPH03x TV Capturer61 60 -> Modular Technology MM100PCTV62 61 -> AG Electronics GMV1 [15cb:0101]63 62 -> Askey CPH061/ BESTBUY Easy TV (bt878)64 63 -> ATI TV-Wonder [1002:0001]65 64 -> ATI TV-Wonder VE [1002:0003]66 65 -> Lifeview FlyVideo 2000S LR9067 66 -> Terratec TValueRadio [153b:1135,153b:ff3b]68 67 -> IODATA GV-BCTV4/PCI [10fc:4050]69 68 -> 3Dfx VoodooTV FM (Euro) [10b4:2637]70 69 -> Active Imaging AIMMS71 70 -> Prolink Pixelview PV-BT878P+ (Rev.4C,8E)72 71 -> Lifeview FlyVideo 98EZ (capture only) LR51 [1851:1851]73 72 -> Prolink Pixelview PV-BT878P+9B (PlayTV Pro rev.9B FM+NICAM) [1554:4011]74 73 -> Sensoray 311 [6000:0311]75 74 -> RemoteVision MX (RV605)76 75 -> Powercolor MTV878/ MTV878R/ MTV878F77 76 -> Canopus WinDVR PCI (COMPAQ Presario 3524JP, 5112JP) [0e11:0079]78 77 -> GrandTec Multi Capture Card (Bt878)79 78 -> Jetway TV/Capture JW-TV878-FBK, Kworld KW-TV878RF [0a01:17de]80 79 -> DSP Design TCVIDEO81 80 -> Hauppauge WinTV PVR [0070:4500]82 81 -> IODATA GV-BCTV5/PCI [10fc:4070,10fc:d018]83 82 -> Osprey 100/150 (878) [0070:ff00]84 83 -> Osprey 100/150 (848)85 84 -> Osprey 101 (848)86 85 -> Osprey 101/15187 86 -> Osprey 101/151 w/ svid88 87 -> Osprey 200/201/250/25189 88 -> Osprey 200/250 [0070:ff01]90 89 -> Osprey 210/220/23091 90 -> Osprey 500 [0070:ff02]92 91 -> Osprey 540 [0070:ff04]93 92 -> Osprey 2000 [0070:ff03]94 93 -> IDS Eagle95 94 -> Pinnacle PCTV Sat [11bd:001c]96 95 -> Formac ProTV II (bt878)97 96 -> MachTV98 97 -> Euresys Picolo99 98 -> ProVideo PV150 [aa00:1460,aa01:1461,aa02:1462,aa03:1463,aa04:1464,aa05:1465,aa06:1466,aa07:1467]100 99 -> AD-TVK503101 100 -> Hercules Smart TV Stereo102 101 -> Pace TV & Radio Card103 102 -> IVC-200 [0000:a155,0001:a155,0002:a155,0003:a155,0100:a155,0101:a155,0102:a155,0103:a155]104 103 -> Grand X-Guard / Trust 814PCI [0304:0102]105 104 -> Nebula Electronics DigiTV [0071:0101]106 105 -> ProVideo PV143 [aa00:1430,aa00:1431,aa00:1432,aa00:1433,aa03:1433]107 106 -> PHYTEC VD-009-X1 MiniDIN (bt878)108 107 -> PHYTEC VD-009-X1 Combi (bt878)109 108 -> PHYTEC VD-009 MiniDIN (bt878)110 109 -> PHYTEC VD-009 Combi (bt878)111 110 -> IVC-100 [ff00:a132]112 111 -> IVC-120G [ff00:a182,ff01:a182,ff02:a182,ff03:a182,ff04:a182,ff05:a182,ff06:a182,ff07:a182,ff08:a182,ff09:a182,ff0a:a182,ff0b:a182,ff0c:a182,ff0d:a182,ff0e:a182,ff0f:a182]113 112 -> pcHDTV HD-2000 TV [7063:2000]114 113 -> Twinhan DST + clones [11bd:0026,1822:0001,270f:fc00,1822:0026]115 114 -> Winfast VC100 [107d:6607]116 115 -> Teppro TEV-560/InterVision IV-560117 116 -> SIMUS GVC1100 [aa6a:82b2]118 117 -> NGS NGSTV+119 118 -> LMLBT4120 119 -> Tekram M205 PRO121 120 -> Conceptronic CONTVFMi122 121 -> Euresys Picolo Tetra [1805:0105,1805:0106,1805:0107,1805:0108]123 122 -> Spirit TV Tuner124 123 -> AVerMedia AVerTV DVB-T 771 [1461:0771]125 124 -> AverMedia AverTV DVB-T 761 [1461:0761]126 125 -> MATRIX Vision Sigma-SQ127 126 -> MATRIX Vision Sigma-SLC128 127 -> APAC Viewcomp 878(AMAX)129 128 -> DViCO FusionHDTV DVB-T Lite [18ac:db10,18ac:db11]130 129 -> V-Gear MyVCD131 130 -> Super TV Tuner132 131 -> Tibet Systems 'Progress DVR' CS16133 132 -> Kodicom 4400R (master)134 133 -> Kodicom 4400R (slave)135 134 -> Adlink RTV24136 135 -> DViCO FusionHDTV 5 Lite [18ac:d500]137 136 -> Acorp Y878F [9511:1540]138 137 -> Conceptronic CTVFMi v2139 138 -> Prolink Pixelview PV-BT878P+ (Rev.2E)140 139 -> Prolink PixelView PlayTV MPEG2 PV-M4900141 140 -> Osprey 440 [0070:ff07]142 141 -> Asound Skyeye PCTV143 142 -> Sabrent TV-FM (bttv version)144 143 -> Hauppauge ImpactVCB (bt878) [0070:13eb]145 144 -> MagicTV146 145 -> SSAI Security Video Interface [4149:5353]147 146 -> SSAI Ultrasound Video Interface [414a:5353]148 147 -> VoodooTV 200 (USA) [121a:3000]149 148 -> DViCO FusionHDTV 2 [dbc0:d200]150 149 -> Typhoon TV-Tuner PCI (50684)Hide Line Numbers * CARDLIST.ivtv * ibmcam.txt * CARDLIST.cx23885 * README.saa7134 * zr364xx.txt * w9968cf.txt * README.ir * README.cpia * et61x251.txt * CARDLIST.tuner * CARDLIST.usbvision * README.pvrusb2 * [ cx88 ] * CARDLIST.cx88 * Zoran * CQcam.txt * ov511.txt * extract_xc3028.pl * cpia2_overview.txt * radiotrack.txt * hauppauge-wintv-cx88-ir.txt * not-in-cx2388x-datasheet.txt * CARDLIST.saa7134 * cafe_ccic * [ video4linux ] * meye.txt * * API.html * CARDLIST.em28xx * README.cpia2 * sn9c102.txt * README.cx88 * stv680.txt * CARDLIST.bttv * [ cx2341x ] * v4lgrab.c * [ bttv ] * se401.txt * zc0301.txt * w9966.txt * lifeview.txt * README.ivtv * *About Kernel Documentation Linux Kernel Contact Linux Resources Linux BlogInformation is copyright its respective author. All material is available from the Linux Kernel Source distributed under a GPL License. This page is provided as a free service by mjmwired.net.

Linux Kernel Documentation :: video4linux : CARDLIST.bttv

Blogged with the Flock Browser

http://www.symons.net.au/16chan_dvr_card/dmesg-nforce3-gigabyte-k8ns-ultra-939-stable.txt

Linux version 2.6.13-1.1526_FC4 (bhcompile@hs20-bc1-6.build.redhat.com) (gcc version 4.0.1 20050727 (Red Hat 4.0.1-5)) #1 Wed Sep 28 19:15:10 EDT 2005BIOS-provided physical RAM map: BIOS-e820: 0000000000000000 - 000000000009f800 (usable) BIOS-e820: 000000000009f800 - 00000000000a0000 (reserved) BIOS-e820: 00000000000f0000 - 0000000000100000 (reserved) BIOS-e820: 0000000000100000 - 000000001fff0000 (usable) BIOS-e820: 000000001fff0000 - 000000001fff3000 (ACPI NVS) BIOS-e820: 000000001fff3000 - 0000000020000000 (ACPI data) BIOS-e820: 00000000fec00000 - 00000000fec01000 (reserved) BIOS-e820: 00000000fee00000 - 00000000fee01000 (reserved) BIOS-e820: 00000000ffff0000 - 0000000100000000 (reserved)0MB HIGHMEM available.511MB LOWMEM available.Using x86 segment limits to approximate NX protectionOn node 0 totalpages: 131056 DMA zone: 4096 pages, LIFO batch:1 Normal zone: 126960 pages, LIFO batch:31 HighMem zone: 0 pages, LIFO batch:1DMI 2.3 present.ACPI: RSDP (v000 Nvidia ) @ 0x000f6d80ACPI: RSDT (v001 Nvidia AWRDACPI 0x42302e31 AWRD 0x01010101) @ 0x1fff3000ACPI: FADT (v001 Nvidia AWRDACPI 0x42302e31 AWRD 0x01010101) @ 0x1fff3040ACPI: MADT (v001 Nvidia AWRDACPI 0x42302e31 AWRD 0x01010101) @ 0x1fff79c0ACPI: DSDT (v001 NVIDIA AWRDACPI 0x00001000 MSFT 0x0100000c) @ 0x00000000ACPI: PM-Timer IO Port: 0x1008Allocating PCI resources starting at 20000000 (gap: 20000000:dec00000)Built 1 zonelistsKernel command line: ro root=LABEL=/ rhgbInitializing CPU#0CPU 0 irqstacks, hard=c04e7000 soft=c04e6000PID hash table entries: 2048 (order: 11, 32768 bytes)Detected 2100.225 MHz processor.Using pmtmr for high-res timesourceConsole: colour VGA+ 80x25Dentry cache hash table entries: 131072 (order: 7, 524288 bytes)Inode-cache hash table entries: 65536 (order: 6, 262144 bytes)Memory: 513264k/524224k available (3084k kernel code, 10364k reserved, 704k data, 176k init, 0k highmem)Checking if this processor honours the WP bit even in supervisor mode... Ok.Calibrating delay using timer specific routine.. 4204.70 BogoMIPS (lpj=8409404)Security Framework v1.0.0 initializedSELinux: Initializing.SELinux: Starting in permissive modeselinux_register_security: Registering secondary module capabilityCapability LSM initialized as secondaryMount-cache hash table entries: 512CPU: After generic identify, caps: 078bfbff e3d3fbff 00000000 00000000 00000001 00000000 00000001CPU: After vendor identify, caps: 078bfbff e3d3fbff 00000000 00000000 00000001 00000000 00000001CPU: L1 I Cache: 64K (64 bytes/line), D cache 64K (64 bytes/line)CPU: L2 Cache: 512K (64 bytes/line)CPU: After all inits, caps: 078bf3ff e3d3fbff 00000000 00000010 00000001 00000000 00000001Intel machine check architecture supported.Intel machine check reporting enabled on CPU#0.mtrr: v2.0 (20020519)CPU: AMD Athlon(tm) 64 Processor 3200+ stepping 00Enabling fast FPU save and restore... done.Enabling unmasked SIMD FPU exception support... done.Checking 'hlt' instruction... OK.ACPI: setting ELCR to 0200 (from 1e20)checking if image is initramfs... it isFreeing initrd memory: 1092k freedNET: Registered protocol family 16ACPI: bus type pci registeredPCI: PCI BIOS revision 2.10 entry at 0xfb560, last bus=3PCI: Using configuration type 1ACPI: Subsystem revision 20050408ACPI: Interpreter enabledACPI: Using PIC for interrupt routingACPI: PCI Root Bridge [PCI0] (0000:00)PCI: Probing PCI hardware (bus 00)ACPI: Assume root bridge [\_SB_.PCI0] segment is 0Boot video device is 0000:01:00.0ACPI: PCI Interrupt Routing Table [\_SB_.PCI0._PRT]ACPI: PCI Interrupt Routing Table [\_SB_.PCI0.HUB0._PRT]ACPI: Power Resource [ISAV] (on)ACPI: PCI Interrupt Routing Table [\_SB_.PCI0.AGPB._PRT]ACPI: PCI Interrupt Link [LNK1] (IRQs 3 4 5 6 7 9 10 *11 12 14 15)ACPI: PCI Interrupt Link [LNK2] (IRQs 3 4 5 6 7 9 *10 11 12 14 15)ACPI: PCI Interrupt Link [LNK3] (IRQs 3 4 5 6 7 9 *10 11 12 14 15)ACPI: PCI Interrupt Link [LNK4] (IRQs 3 4 *5 6 7 9 10 11 12 14 15)ACPI: PCI Interrupt Link [LNK5] (IRQs 3 4 5 6 7 *9 10 11 12 14 15)ACPI: PCI Interrupt Link [LUBA] (IRQs 3 4 *5 6 7 9 10 11 12 14 15)ACPI: PCI Interrupt Link [LUBB] (IRQs 3 4 *5 6 7 9 10 11 12 14 15)ACPI: PCI Interrupt Link [LMAC] (IRQs 3 4 5 6 7 9 10 11 *12 14 15)ACPI: PCI Interrupt Link [LAPU] (IRQs 3 4 5 6 7 9 10 11 12 14 15) *0, disabled.ACPI: PCI Interrupt Link [LACI] (IRQs 3 4 5 6 7 9 10 11 *12 14 15)ACPI: PCI Interrupt Link [LMCI] (IRQs 3 4 5 6 7 9 10 11 12 14 15) *0, disabled.ACPI: PCI Interrupt Link [LSMB] (IRQs 3 4 5 6 7 9 10 11 *12 14 15)ACPI: PCI Interrupt Link [LUB2] (IRQs 3 4 *5 6 7 9 10 11 12 14 15)ACPI: PCI Interrupt Link [LFIR] (IRQs 3 4 5 6 7 9 10 11 12 14 15) *0, disabled.ACPI: PCI Interrupt Link [L3CM] (IRQs 3 4 5 6 7 9 10 11 12 14 15) *0, disabled.ACPI: PCI Interrupt Link [LIDE] (IRQs 3 4 5 6 7 9 10 11 12 14 15) *0, disabled.ACPI: PCI Interrupt Link [LSID] (IRQs 3 4 5 6 7 9 10 11 12 14 15) *0, disabled.ACPI: PCI Interrupt Link [LFID] (IRQs 3 4 5 6 7 9 10 *11 12 14 15)ACPI: PCI Interrupt Link [APC1] (IRQs *16), disabled.ACPI: PCI Interrupt Link [APC2] (IRQs *17), disabled.ACPI: PCI Interrupt Link [APC3] (IRQs *18), disabled.ACPI: PCI Interrupt Link [APC4] (IRQs *19), disabled.ACPI: PCI Interrupt Link [APC5] (IRQs *16), disabled.ACPI: PCI Interrupt Link [APCF] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [APCG] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [APCH] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [APCI] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [APCJ] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [APCK] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [APCS] (IRQs *23), disabled.ACPI: PCI Interrupt Link [APCL] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [APCM] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [AP3C] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [APCZ] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [APSI] (IRQs 20 21 22) *0, disabled.ACPI: PCI Interrupt Link [APSJ] (IRQs 20 21 22) *0, disabled.Linux Plug and Play Support v0.97 (c) Adam Belaypnp: PnP ACPI initpnp: PnP ACPI: found 12 devicesusbcore: registered new driver usbfsusbcore: registered new driver hubPCI: Using ACPI for IRQ routingPCI: If a device doesn't work, try "pci=routeirq". If it helps, post a reportpnp: 00:00: ioport range 0x1000-0x107f could not be reservedpnp: 00:00: ioport range 0x1080-0x10ff has been reservedpnp: 00:00: ioport range 0x1400-0x147f has been reservedpnp: 00:00: ioport range 0x1480-0x14ff could not be reservedpnp: 00:00: ioport range 0x1800-0x187f has been reservedpnp: 00:00: ioport range 0x1880-0x18ff has been reservedPCI: Bridge: 0000:00:0b.0 IO window: disabled. MEM window: f2000000-f3ffffff PREFETCH window: f0000000-f1ffffffPCI: Bridge: 0000:02:06.0 IO window: disabled. MEM window: disabled. PREFETCH window: f6000000-f6ffffffPCI: Bridge: 0000:00:0e.0 IO window: 9000-afff MEM window: f4000000-f5ffffff PREFETCH window: f6000000-f7ffffffPCI: Setting latency timer of device 0000:00:0e.0 to 64apm: BIOS version 1.2 Flags 0x07 (Driver version 1.16ac)apm: overridden by ACPI.audit: initializing netlink socket (disabled)audit(1129167859.200:1): initializedTotal HugeTLB memory allocated, 0VFS: Disk quotas dquot_6.5.1Dquot-cache hash table entries: 1024 (order 0, 4096 bytes)SELinux: Registering netfilter hooksInitializing Cryptographic APIksign: Installing public key dataLoading keyring- Added public key 54AD4C348BA3456- User ID: Red Hat, Inc. (Kernel Module GPG key)pci_hotplug: PCI Hot Plug PCI Core version: 0.5ACPI: CPU0 (power states: C1[C1])isapnp: Scanning for PnP cards...isapnp: No Plug & Play device foundReal Time Clock Driver v1.12Linux agpgart interface v0.101 (c) Dave Jonesagpgart: Detected AGP bridge 0agpgart: Setting up Nforce3 AGP.agpgart: AGP aperture is 128M @ 0xe8000000PNP: PS/2 controller doesn't have AUX irq; using default 0xcPNP: PS/2 Controller [PNP0303:PS2K] at 0x60,0x64 irq 112serio: i8042 AUX port at 0x60,0x64 irq 12serio: i8042 KBD port at 0x60,0x64 irq 1Serial: 8250/16550 driver $Revision: 1.90 $ 32 ports, IRQ sharing enabledttyS0 at I/O 0x3f8 (irq = 4) is a 16550AttyS1 at I/O 0x2f8 (irq = 3) is a 16550AttyS0 at I/O 0x3f8 (irq = 4) is a 16550AttyS1 at I/O 0x2f8 (irq = 3) is a 16550Aio scheduler noop registeredio scheduler anticipatory registeredio scheduler deadline registeredio scheduler cfq registeredRAMDISK driver initialized: 16 RAM disks of 16384K size 1024 blocksizeUniform Multi-Platform E-IDE driver Revision: 7.00alpha2ide: Assuming 33MHz system bus speed for PIO modes; override with idebus=xxNFORCE3-250: IDE controller at PCI slot 0000:00:08.0NFORCE3-250: chipset revision 162NFORCE3-250: not 100% native mode: will probe irqs laterNFORCE3-250: BIOS didn't set cable bits correctly. Enabling workaround.NFORCE3-250: BIOS didn't set cable bits correctly. Enabling workaround.NFORCE3-250: 0000:00:08.0 (rev a2) UDMA133 controller ide0: BM-DMA at 0xf000-0xf007, BIOS settings: hda:DMA, hdb:DMA ide1: BM-DMA at 0xf008-0xf00f, BIOS settings: hdc:DMA, hdd:DMAProbing IDE interface ide0...hda: WDC WD2000JB-00GVC0, ATA DISK drivehdb: WDC WD2000JB-00GVA0, ATA DISK driveide0 at 0x1f0-0x1f7,0x3f6 on irq 14Probing IDE interface ide1...hdc: WDC WD800BB-75CAA0, ATA DISK driveide1 at 0x170-0x177,0x376 on irq 15hda: max request size: 1024KiBhda: 390721968 sectors (200049 MB) w/8192KiB Cache, CHS=24321/255/63, UDMA(100)hda: cache flushes supported hda: hda1 hda2 hda3 hda4hdb: max request size: 1024KiBhdb: 390721968 sectors (200049 MB) w/8192KiB Cache, CHS=24321/255/63, UDMA(100)hdb: cache flushes supported hdb: hdb1hdc: max request size: 128KiBhdc: Host Protected Area detected. current capacity is 156250000 sectors (80000 MB) native capacity is 156301488 sectors (80026 MB)hdc: Host Protected Area disabled.hdc: 156301488 sectors (80026 MB) w/2048KiB Cache, CHS=65535/16/63, UDMA(100)hdc: cache flushes not supported hdc: hdc1ide-floppy driver 0.99.newideusbcore: registered new driver hiddevusbcore: registered new driver usbhiddrivers/usb/input/hid-core.c: v2.01:USB HID core drivermice: PS/2 mouse device common for all micemd: md driver 0.90.2 MAX_MD_DEVS=256, MD_SB_DISKS=27md: bitmap version 3.38NET: Registered protocol family 2IP route cache hash table entries: 8192 (order: 3, 32768 bytes)TCP established hash table entries: 32768 (order: 6, 262144 bytes)TCP bind hash table entries: 32768 (order: 7, 917504 bytes)TCP: Hash tables configured (established 32768 bind 32768)TCP reno registeredTCP bic registeredInitializing IPsec netlink socketNET: Registered protocol family 1NET: Registered protocol family 17Using IPI Shortcut modeACPI wakeup devices: HUB0 HUB1 USB0 USB1 USB2 F139 MMAC MMCI UAR1 ACPI: (supports S0 S1 S4 S5)Freeing unused kernel memory: 176k freedkjournald starting. Commit interval 5 secondsEXT3-fs: mounted filesystem with ordered data mode.input: AT Translated Set 2 keyboard on isa0060/serio0SELinux: Disabled at runtime.SELinux: Unregistering netfilter hooksSCSI subsystem initializedlibata version 1.12 loaded.sata_nv version 0.6ACPI: PCI Interrupt Link [LFID] enabled at IRQ 11PCI: setting IRQ 11 as level-triggeredACPI: PCI Interrupt 0000:00:0a.0[A] -> Link [LFID] -> GSI 11 (level, low) -> IRQ 11PCI: Setting latency timer of device 0000:00:0a.0 to 64ata1: SATA max UDMA/133 cmd 0x9F0 ctl 0xBF2 bmdma 0xDC00 irq 11ata2: SATA max UDMA/133 cmd 0x970 ctl 0xB72 bmdma 0xDC08 irq 11ata1: no device found (phy stat 00000000)scsi0 : sata_nvata2: no device found (phy stat 00000000)scsi1 : sata_nvsata_sil version 0.9ACPI: PCI Interrupt Link [LNK2] enabled at IRQ 10PCI: setting IRQ 10 as level-triggeredACPI: PCI Interrupt 0000:02:0d.0[A] -> Link [LNK2] -> GSI 10 (level, low) -> IRQ 10ata3: SATA max UDMA/100 cmd 0xE0838080 ctl 0xE083808A bmdma 0xE0838000 irq 10ata4: SATA max UDMA/100 cmd 0xE08380C0 ctl 0xE08380CA bmdma 0xE0838008 irq 10ata3: no device found (phy stat 00000000)scsi2 : sata_silata4: no device found (phy stat 00000000)scsi3 : sata_silfloppy0: no floppy controllers foundACPI: PCI Interrupt Link [LNK4] enabled at IRQ 5PCI: setting IRQ 5 as level-triggeredACPI: PCI Interrupt 0000:02:0b.0[A] -> Link [LNK4] -> GSI 5 (level, low) -> IRQ 5ACPI: PCI Interrupt 0000:02:0b.0[A] -> Link [LNK4] -> GSI 5 (level, low) -> IRQ 5eth0: Yukon Gigabit Ethernet 10/100/1000Base-T Adapter PrefPort:A RlmtMode:Check Link StateACPI: PCI Interrupt Link [LACI] enabled at IRQ 12PCI: setting IRQ 12 as level-triggeredACPI: PCI Interrupt 0000:00:06.0[A] -> Link [LACI] -> GSI 12 (level, low) -> IRQ 12PCI: Setting latency timer of device 0000:00:06.0 to 64intel8x0_measure_ac97_clock: measured 54815 usecsintel8x0: clocking to 46984shpchp: shpc_init : shpc_cap_offset == 0shpchp: shpc_init : shpc_cap_offset == 0shpchp: shpc_init : shpc_cap_offset == 0shpchp: Standard Hot Plug PCI Controller Driver version: 0.4forcedeth.c: Reverse Engineered nForce ethernet driver. Version 0.35.ACPI: PCI Interrupt Link [LMAC] enabled at IRQ 12ACPI: PCI Interrupt 0000:00:05.0[A] -> Link [LMAC] -> GSI 12 (level, low) -> IRQ 12PCI: Setting latency timer of device 0000:00:05.0 to 64eth1: forcedeth.c: subsystem: 01458:e000 bound to 0000:00:05.0i2c_core: Unknown parameter `i2c_debug'i2c_adapter i2c-0: nForce2 SMBus adapter at 0x1c00i2c_adapter i2c-1: nForce2 SMBus adapter at 0x2000Linux video capture interface: v1.00bttv: driver version 0.9.16 loadedbttv: using 8 buffers with 2080k (520 pages) each for capturebttv: Bt8xx card found (0).ACPI: PCI Interrupt 0000:02:07.0[A] -> Link [LNK4] -> GSI 5 (level, low) -> IRQ 5bttv0: Bt878 (rev 17) at 0000:02:07.0, irq: 5, latency: 32, mmio: 0xf7000000bttv0: using: Prolink Pixelview PV-BT878P+ (Rev.4C,8E) [card=70,insmod option]bttv0: gpio: en=00000000, out=00000000 in=00f36000 [init]i2c-algo-bit.o: (0) scl=0, sda=0i2c-algo-bit.o: bt878 #0 [sw] seems to be busy.bttv0: using tuner=25tvaudio: TV audio decoder + audio/video mux drivertvaudio: known chips: tda9840,tda9873h,tda9874h/a,tda9850,tda9855,tea6300,tea6320,tea6420,tda8425,pic16c54 (PV951),ta8874zbttv0: registered device video0bttv0: registered device vbi0bttv0: PLL: 28636363 => 35468950 .. okbttv0: add subdevice "remote0"bttv: Bt8xx card found (1).ACPI: PCI Interrupt Link [LNK3] enabled at IRQ 10ACPI: PCI Interrupt 0000:03:0c.0[A] -> Link [LNK3] -> GSI 10 (level, low) -> IRQ 10bttv1: Bt878 (rev 17) at 0000:03:0c.0, irq: 10, latency: 32, mmio: 0xf6000000bttv1: using: Tibet Systems 'Progress DVR' CS16 [card=131,insmod option]bttv1: gpio: en=00000000, out=00000000 in=008fffff [init]i2c-algo-bit.o: (0) scl=1, sda=1i2c-algo-bit.o: (1) scl=1, sda=0i2c-algo-bit.o: (2) scl=1, sda=1i2c-algo-bit.o: (3) scl=0, sda=1i2c-algo-bit.o: (4) scl=1, sda=1i2c-algo-bit.o: bt878 #1 [sw] passed test.bttv1: using tuner=-1bttv1: i2c: checking for TDA9887 @ 0x86... not foundbttv1: registered device video1bttv1: registered device vbi1bttv1: PLL: 28636363 => 35468950 .. okbttv: Bt8xx card found (2).ACPI: PCI Interrupt 0000:03:0d.0[A] -> Link [LNK4] -> GSI 5 (level, low) -> IRQ 5bttv2: Bt878 (rev 17) at 0000:03:0d.0, irq: 5, latency: 32, mmio: 0xf6002000bttv2: using: Tibet Systems 'Progress DVR' CS16 [card=131,insmod option]bttv2: gpio: en=00000000, out=00000000 in=009fffff [init]i2c-algo-bit.o: (0) scl=1, sda=1i2c-algo-bit.o: (1) scl=1, sda=0i2c-algo-bit.o: (2) scl=1, sda=1i2c-algo-bit.o: (3) scl=0, sda=1i2c-algo-bit.o: (4) scl=1, sda=1i2c-algo-bit.o: bt878 #2 [sw] passed test.bttv2: using tuner=-1bttv2: i2c: checking for TDA9887 @ 0x86... not foundbttv2: registered device video2bttv2: registered device vbi2bttv2: PLL: 28636363 => 35468950 .. okbttv: Bt8xx card found (3).ACPI: PCI Interrupt Link [LNK1] enabled at IRQ 11ACPI: PCI Interrupt 0000:03:0e.0[A] -> Link [LNK1] -> GSI 11 (level, low) -> IRQ 11bttv3: Bt878 (rev 17) at 0000:03:0e.0, irq: 11, latency: 32, mmio: 0xf6004000bttv3: using: Tibet Systems 'Progress DVR' CS16 [card=131,insmod option]bttv3: gpio: en=00000000, out=00000000 in=00af7fff [init]i2c-algo-bit.o: (0) scl=1, sda=1i2c-algo-bit.o: (1) scl=1, sda=0i2c-algo-bit.o: (2) scl=1, sda=1i2c-algo-bit.o: (3) scl=0, sda=1i2c-algo-bit.o: (4) scl=1, sda=1i2c-algo-bit.o: bt878 #3 [sw] passed test.bttv3: using tuner=-1bttv3: i2c: checking for TDA9887 @ 0x86... not foundbttv3: registered device video3bttv3: registered device vbi3bttv3: PLL: 28636363 => 35468950 .. okbttv: Bt8xx card found (4).ACPI: PCI Interrupt 0000:03:0f.0[A] -> Link [LNK2] -> GSI 10 (level, low) -> IRQ 10bttv4: Bt878 (rev 17) at 0000:03:0f.0, irq: 10, latency: 32, mmio: 0xf6006000bttv4: using: Tibet Systems 'Progress DVR' CS16 [card=131,insmod option]bttv4: gpio: en=00000000, out=00000000 in=00bff5ff [init]i2c-algo-bit.o: (0) scl=1, sda=1i2c-algo-bit.o: (1) scl=1, sda=0i2c-algo-bit.o: (2) scl=1, sda=1i2c-algo-bit.o: (3) scl=0, sda=1i2c-algo-bit.o: (4) scl=1, sda=1i2c-algo-bit.o: bt878 #4 [sw] passed test.bttv4: using tuner=-1bttv4: i2c: checking for TDA9887 @ 0x86... not foundbttv4: registered device video4bttv4: registered device vbi4bttv4: PLL: 28636363 => 35468950 .. okbt878: AUDIO driver version 0.0.0 loadedbt878: Bt878 AUDIO function found (0).ACPI: PCI Interrupt 0000:02:07.1[A] -> Link [LNK4] -> GSI 5 (level, low) -> IRQ 5bt878(0): Bt878 (rev 17) at 02:07.1, irq: 5, latency: 32, memory: 0xf7001000bt878: Bt878 AUDIO function found (1).ACPI: PCI Interrupt 0000:03:0c.1[A] -> Link [LNK3] -> GSI 10 (level, low) -> IRQ 10bt878(1): Bt878 (rev 17) at 03:0c.1, irq: 10, latency: 32, memory: 0xf6001000bt878: Bt878 AUDIO function found (2).ACPI: PCI Interrupt 0000:03:0d.1[A] -> Link [LNK4] -> GSI 5 (level, low) -> IRQ 5bt878(2): Bt878 (rev 17) at 03:0d.1, irq: 5, latency: 32, memory: 0xf6003000bt878: Bt878 AUDIO function found (3).ACPI: PCI Interrupt 0000:03:0e.1[A] -> Link [LNK1] -> GSI 11 (level, low) -> IRQ 11bt878(3): Bt878 (rev 17) at 03:0e.1, irq: 11, latency: 32, memory: 0xf6005000bt878: Bt878 AUDIO function found (4).ACPI: PCI Interrupt 0000:03:0f.1[A] -> Link [LNK2] -> GSI 10 (level, low) -> IRQ 10bt878(4): Bt878 (rev 17) at 03:0f.1, irq: 10, latency: 32, memory: 0xf6007000ACPI: PCI Interrupt Link [LUB2] enabled at IRQ 5ACPI: PCI Interrupt 0000:00:02.2[C] -> Link [LUB2] -> GSI 5 (level, low) -> IRQ 5PCI: Setting latency timer of device 0000:00:02.2 to 64ehci_hcd 0000:00:02.2: EHCI Host Controllerehci_hcd 0000:00:02.2: debug port 1ehci_hcd 0000:00:02.2: new USB bus registered, assigned bus number 1ehci_hcd 0000:00:02.2: irq 5, io mem 0xf8004000PCI: cache line size of 64 is not supported by device 0000:00:02.2ehci_hcd 0000:00:02.2: USB 2.0 initialized, EHCI 1.00, driver 10 Dec 2004hub 1-0:1.0: USB hub foundhub 1-0:1.0: 8 ports detectedohci_hcd: 2005 April 22 USB 1.1 'Open' Host Controller (OHCI) Driver (PCI)ACPI: PCI Interrupt Link [LUBA] enabled at IRQ 5ACPI: PCI Interrupt 0000:00:02.0[A] -> Link [LUBA] -> GSI 5 (level, low) -> IRQ 5PCI: Setting latency timer of device 0000:00:02.0 to 64ohci_hcd 0000:00:02.0: OHCI Host Controllerohci_hcd 0000:00:02.0: new USB bus registered, assigned bus number 2ohci_hcd 0000:00:02.0: irq 5, io mem 0xf8002000hub 2-0:1.0: USB hub foundhub 2-0:1.0: 4 ports detectedACPI: PCI Interrupt Link [LUBB] enabled at IRQ 5ACPI: PCI Interrupt 0000:00:02.1[B] -> Link [LUBB] -> GSI 5 (level, low) -> IRQ 5PCI: Setting latency timer of device 0000:00:02.1 to 64ohci_hcd 0000:00:02.1: OHCI Host Controllerohci_hcd 0000:00:02.1: new USB bus registered, assigned bus number 3ohci_hcd 0000:00:02.1: irq 5, io mem 0xf8003000hub 3-0:1.0: USB hub foundhub 3-0:1.0: 4 ports detectedieee1394: Initialized config rom entry `ip1394'ohci1394: $Rev: 1299 $ Ben Collins ACPI: PCI Interrupt 0000:02:0e.0[A] -> Link [LNK3] -> GSI 10 (level, low) -> IRQ 10ohci1394: fw-host0: OHCI-1394 1.1 (PCI): IRQ=[10] MMIO=[f5008000-f50087ff] Max Packet=[4096]ieee1394: Host added: ID:BUS[0-00:1023] GUID[00148556000cd46a]ACPI: Power Button (FF) [PWRF]ACPI: Power Button (CM) [PWRB]ibm_acpi: ec object not foundmd: Autodetecting RAID arrays.md: autorun ...md: ... autorun DONE.device-mapper: 4.4.0-ioctl (2005-01-12) initialised: dm-devel@redhat.comEXT3 FS on hda3, internal journalkjournald starting. Commit interval 5 secondsEXT3 FS on hda1, internal journalEXT3-fs: mounted filesystem with ordered data mode.SGI XFS with ACLs, security attributes, large block numbers, no debug enabledSGI XFS Quota Management subsystemXFS mounting filesystem hdb1Ending clean XFS mount for filesystem: hdb1ReiserFS: hda4: found reiserfs format "3.6" with standard journalReiserFS: hda4: using ordered data modeReiserFS: hda4: journal params: device hda4, size 8192, journal first block 18, max trans len 1024, max batch 900, max commit age 30, max trans age 30ReiserFS: hda4: checking transaction log (hda4)ReiserFS: hda4: Using r5 hash to sort namesAdding 1004052k swap on /dev/hda2. Priority:-1 extents:1

Blogged with the Flock Browser

The phytocannabinoid Delta(9)-tetrahydrocannabivarin modulates inhibitory neurotransmission in the cerebellum.

The phytocannabinoid Delta(9)-tetrahydrocannabivarin modulates inhibitory neurotransmission in the cerebellum.

Br J Pharmacol. 2008 Mar 3;

Authors: Ma YL, Weston SE, Whalley BJ, Stephens GJ

Background and purposeThe phytocannabinoid Delta(9)-tetrahydrocannabivarin (Delta(9)-THCV) has been reported to exhibit a diverse pharmacology; here, we investigate functional effects of Delta(9)-THCV, extracted from Cannabis sativa, using electrophysiological techniques to define its mechanism of action in the CNS.Experimental approachEffects of Delta(9)-THCV and synthetic cannabinoid agents on inhibitory neurotransmission at interneurone-Purkinje cell (IN-PC) synapses were correlated with effects on spontaneous PC output using single-cell and multi-electrode array (MEA) electrophysiological recordings respectively, in mouse cerebellar brain slices in vitro.Key resultsThe cannabinoid receptor agonist WIN 55,212-2 (WIN55) decreased miniature inhibitory postsynaptic current (mIPSC) frequency at IN-PC synapses. WIN55-induced inhibition was reversed by Delta(9)-THCV, and also by the CB(1) receptor antagonist AM251; Delta(9)-THCV or AM251 acted to increase mIPSC frequency beyond basal values. When applied alone, Delta(9)-THCV, AM251 or rimonabant increased mIPSC frequency. Pre-incubation with Delta(9)-THCV blocked WIN55-induced inhibition. In MEA recordings, WIN55 increased PC spike firing rate; Delta(9)-THCV and AM251 acted in the opposite direction to decrease spike firing. The effects of Delta(9)-THCV and WIN55 were attenuated by the GABA(A) receptor antagonist bicuculline methiodide.Conclusions and implicationsWe show for the first time that Delta(9)-THCV acts as a functional CB(1) receptor antagonist in the CNS to modulate inhibitory neurotransmission at IN-PC synapses and spontaneous PC output. Delta(9)-THCV- and AM251-induced increases in mIPSC frequency beyond basal levels were consistent with basal CB(1) receptor activity. WIN55-induced increases in PC spike firing rate were consistent with synaptic disinhibition; whilst Delta(9)-THCV- and AM251-induced decreases in spike firing suggest a mechanism of PC inhibition.British Journal of Pharmacology advance online publication, 3 March 2008; doi:10.1038/bjp.2008.57.

PMID: 18311186 [PubMed - as supplied by publisher]

View Original Article

Blogged with the Flock Browser

Reciprocal inhibition of G-protein signaling is induced by CB(1) cannabinoid and GABA(B) receptor interactions in rat hippocampal membranes.

Reciprocal inhibition of G-protein signaling is induced by CB(1) cannabinoid and GABA(B) receptor interactions in rat hippocampal membranes.

Neurochem Int. 2008 Feb 29;

Authors: Cinar R, Freund TF, Katona I, Mackie K, Szucs M

Cannabinoid CB(1) and the metabotropic GABA(B) receptors have been shown to display similar pharmacological effects and co-localization in certain brain regions. Previous studies have reported a functional link between the two systems. As a first step to investigate the underlying molecular mechanism, here we show cross-inhibition of G-protein signaling between GABA(B) and CB(1) receptors in rat hippocampal membranes. The CB(1) agonist R-Win55,212-2 displayed high potency and efficacy in stimulating guanosine-5'-O-(3-[(35)S]thio)triphosphate, [(35)S]GTPgammaS binding. Its effect was completely blocked by the specific CB(1) antagonist AM251 suggesting that the signaling was via CB(1) receptors. The GABA(B) agonists baclofen and SKF97541 also elevated [(35)S]GTPgammaS binding by about 60%, with potency values in the micromolar range. Phaclofen behaved as a low potency antagonist with an ED(50) approximately 1mM. However, phaclofen at low doses (1 and 10nM) slightly but significantly attenuated maximal stimulation of [(35)S]GTPgammaS binding by the CB(1) agonist R-Win55,212-2. The observation that higher concentrations of phaclofen had no such effect rule out the possibility of its direct action on CB(1) receptors. The pharmacologically inactive stereoisomer S-Win55,212-3 had no effect either alone or in combination with phaclofen establishing that the interaction is stereospecific in hippocampus. The specific CB(1) antagonist AM251 at a low dose (1nM) also inhibited the efficacy of G-protein signaling of the GABA(B) receptor agonist SKF97541. Cross-talk of the two receptor systems was not detected in either spinal cord or cerebral cortex membranes. It is speculated that the interaction might occur via an allosteric interaction between a subset of GABA(B) and CB(1) receptors in rat hippocampal membranes. Although the exact molecular mechanism of the reciprocal inhibition between CB(1) and GABA(B) receptors will have to be explored by future studies it is intriguing that the cross-talk might be involved in balance tuning the endocannabinoid and GABAergic signaling in hippocampus.

PMID: 18407377 [PubMed - as supplied by publisher]

View Original Article

Blogged with the Flock Browser

Thursday 13 November 2008

Monday 10 November 2008

Wednesday 5 November 2008

Thursday 16 October 2008

Monday 13 October 2008

Friday 10 October 2008

Friday 3 October 2008

Wednesday 1 October 2008

Wednesday 6 August 2008

Wednesday 2 July 2008

Thursday 29 May 2008

Thursday 22 May 2008

Wednesday 16 April 2008

Blog Archive

About Me